An infectious clone of the West Nile flavivirus.
Analysis of disulfides present in the membrane proteins of the West Nile flavivirus.
Cleavage preference distinguishes the two-component NS2B-NS3 serine proteinases of Dengue and West Nile viruses.
Comparative Flavivirus-Host Protein Interaction Mapping Reveals Mechanisms of Dengue and Zika Virus Pathogenesis.
Human Sec3 protein is a novel transcriptional and translational repressor of flavivirus.
Infectious entry of West Nile virus occurs through a clathrin-mediated endocytic pathway.
Nuclear localization of flavivirus RNA synthesis in infected cells.
Primary structure of the West Nile flavivirus genome region coding for all nonstructural proteins.
Role of PDZ-binding motif from West Nile virus NS5 protein on viral replication.
Sequence analysis of the membrane protein V3 of the flavivirus West Nile virus and of its gene.
Sequence analysis of the viral core protein and the membrane-associated proteins V1 and NV2 of the flavivirus West Nile virus and of the genome sequence for these proteins.
Structural basis for the activation of flaviviral NS3 proteases from dengue and West Nile virus.
Structural basis for the preferential recognition of immature flaviviruses by a fusion-loop antibody.
Structural evidence for regulation and specificity of flaviviral proteases and evolution of the Flaviviridae fold.
Structure and function of flavivirus NS5 methyltransferase.
Studies on the glycosylation of flavivirus E proteins and the role of carbohydrate in antigenic structure.
West Nile virus and dengue virus capsid protein negates the antiviral activity of human Sec3 protein through the proteasome pathway.